chr11-2572862-T-C
Variant summary
The NM_000218.3(KCNQ1):c.797T>C (p.Leu266Pro) variant causes a missense change involving the alteration of a non-conserved nucleotide. The variant allele was found at a cumulative frequency of 0.0000089 (AC=13) in the gnomAD database across 1,461,486 control chromosomes (no homozygotes observed). The grpmax filtering allele frequency (95% CI) is 0.00000575. In-silico predictor (REVEL) classifies this variant as likely damaging/oncogenic. Splicing prediction tools (SpliceAI) predict no significant impact on normal splicing. Variant has been reported in ClinVar as Pathogenic/Likely Pathogenic (★★). ClinVar reports functional evidence for this variant: "SCV000074144: Experimental studies have shown that this missense change affects KCNQ1 function (PMID:21451124)." and additional evidence is available in ClinVar. A different missense at the same amino acid position has been reported as Pathogenic/Likely Pathogenic in ClinVar: p.L266Q: Likely_pathogenic (ClinVar VariationId 2498480, 1 star) This exact variant is curated in the UniProt human variants database as Uncertain Significance.
Frequency
Consequence
NM_000218.3 missense
Scores
Clinical Significance
Conservation
Publications
- long QT syndromeInheritance: AD Classification: DEFINITIVE Submitted by: ClinGen
- Jervell and Lange-Nielsen syndromeInheritance: AR Classification: DEFINITIVE Submitted by: ClinGen
- Jervell and Lange-Nielsen syndrome 1Inheritance: AR Classification: DEFINITIVE, STRONG Submitted by: G2P, Labcorp Genetics (formerly Invitae)
- long QT syndrome 1Inheritance: AR, AD Classification: DEFINITIVE, STRONG Submitted by: G2P, Labcorp Genetics (formerly Invitae)
- atrial fibrillation, familial, 3Inheritance: AD Classification: STRONG Submitted by: Labcorp Genetics (formerly Invitae)
- short QT syndromeInheritance: AD Classification: STRONG, SUPPORTIVE Submitted by: ClinGen, Orphanet
- short QT syndrome type 2Inheritance: AD Classification: STRONG Submitted by: G2P, Labcorp Genetics (formerly Invitae)
- familial atrial fibrillationInheritance: AD Classification: SUPPORTIVE Submitted by: Orphanet
- Jervell and Lange-Nielsen syndromeInheritance: AR Classification: SUPPORTIVE Submitted by: Orphanet
- familial hypertrophic cardiomyopathyInheritance: AD Classification: NO_KNOWN Submitted by: G2P
- hypertrophic cardiomyopathyInheritance: AD Classification: NO_KNOWN Submitted by: ClinGen
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Classification according to ACMG Germline Pathogenicity v2019
Our verdict: Pathogenic. The variant received 18 points.
| Apply | Criterion Code & Title / Strength | Rationale / Clinical Context | |
|---|---|---|---|
| PS3Functional studies show damaging effect | Well-established functional study supports damaging effect (PS3); SCV000074144: Experimental studies have shown that this missense change affects KCNQ1 function (PMID: 21451124).; SCV004030095: "At least one publication reports experimental evidence evaluating an impact on protein function demonstrating an effect on channel function (Jons_2011)." PMID:26743238; SCV004827548: A functional study has shown that the variant causes significant potassium current changes in a transfected Xenopus oocyte model (PMID: 21451124).; SCV000234419: Published functional studies demonstrate a damaging effect on channel function (Jons et al., 2011); SCV004565333: Functional studies show this variant has an effect on channel function (Jons 2011). PMID: 21451124; SCV000320565: "In an in vitro study, this variant disrupted channel function, suggesting an association with increased risk for cardiac events (Jons, 2011)."; SCV006061192: A functional study has shown that the variant causes significant potassium current changes in a transfected Xenopus oocyte model (PMID: 21451124). | ||
| PM1Mutational hot spot or critical domain | Missense neighbourhood hotspot (≥2 P/LP within ±8 AA, OR ≥ 5 vs. background) — PM1; In-domain: 0 pathogenic, 0 benign rare missense variants.; ±8 AA neighbourhood: 27 pathogenic, 1 benign (OR vs. background: 91.7). | ||
| PM2Absent/extremely low in population databases | Very rare in gnomAD for AD/XL gene (popmax AF < threshold/10) — PM2; GnomAD popmax AF = 0.00000575 — very rare for AD+AR gene (base 0.0001, raised to 0.00022 by highest known P/LP ClinVar AF 0.00022) — PM2 moderate. | ||
| PM5Novel missense at known pathogenic residue | Different pathogenic missense at same AA residue in ClinVar (PM5); ClinVar contains a germline Pathogenic/Likely Pathogenic entry at the same amino acid position with a different amino acid change: p.L266Q: Likely_pathogenic (ClinVar VariationId 2498480, 1 star) | ||
| PP3Computational evidence supports damaging effect | Germline meta computational scorer (REVEL/MetaRNN/BayesDel) predicts damaging effect — strong evidence (PP3); Splicing verdict: not pathogenic.; Germline computational verdict: pathogenic (Strong). | ||
| PP5Reputable source reports pathogenic | ClinVar 2-star pathogenic — strong (PP5); ClinVar submissions overwhelmingly pathogenic (≥10 total, ≥80% P/LP, <10% B/LB) — strong (PP5, count-based); ClinVar germline classification: Pathogenic/Likely Pathogenic, 2 star(s). ClinVar submissions strongly and reliably favor pathogenicity (15/15 total P/LP). |
Variant Effect in Transcripts
Automated classification analysis was done for transcript: NM_000218.3. You can select a different transcript below to see updated classification assignments.
RefSeq Transcripts
| Sel. | Gene / Transcript | Tags | HGVSc / HGVSp | Effect | Exon rank | Protein / UniProt |
|---|---|---|---|---|---|---|
KCNQ1 | MANE Select | c.797T>C p.Leu266Pro | missense | Exon 6 of 16 | Prot: NP_000209.2 | |
KCNQ1 | c.797T>C p.Leu266Pro | missense | Exon 6 of 15 | Prot: NP_001393765.1 | ||
KCNQ1 | c.527T>C p.Leu176Pro | missense | Exon 7 of 17 | Prot: NP_001393766.1 |
Ensembl Transcripts
| Sel. | Gene / Transcript | Tags | HGVSc / HGVSp | Effect | Exon rank | Protein / UniProt |
|---|---|---|---|---|---|---|
KCNQ1 | TSL:1MANE Select | c.797T>C p.Leu266Pro | missense | Exon 6 of 16 | Prot: ENSP00000155840.2 UniProt: | |
KCNQ1 | TSL:1 | c.416T>C p.Leu139Pro | missense | Exon 6 of 16 | Prot: ENSP00000334497.5 UniProt: | |
KCNQ1 | c.794T>C p.Leu265Pro | missense | Exon 6 of 16 | Prot: ENSP00000581056.1 UniProt: |
Frequencies
Allele frequencies (AF), counts (AC/AN), homozygotes and coverage
| Source / population | AF | AC | Hom | AN | Coverage |
|---|---|---|---|---|---|
Global population databases 5 sources | |||||
GnomAD3 genomes | Cov:33 | ||||
GnomAD4 exome | AF: 0.00000890 | AC:13 | Hom:0 | AN:1461486 | Cov:32 |
GnomAD4 genome | Cov:33 | ||||
TOPMed (Bravo) | AF: 0.0000113 | AC:3 | Hom:0 | AN:264690 | |
ALFA (dbGaP/dbSNP Allele Frequency Aggregator) | AF: 0.0000128 | AC:3 | Hom:0 | AN:233892 | |
ClinVar
Computational Scores
| Algorithm | Calibrated pred. | Score | Prediction |
|---|---|---|---|
AlphaGenome AVI | Pathogenic | 26 | |
AlphaMissense | Pathogenic | 1.0 | |
BayesDel_addAF | Pathogenic | 0.54 | D |
BayesDel_noAF | Pathogenic | 0.54 | |
CADD | Pathogenic | 30 | |
CardioboostArm | Pathogenic | 1.0 | |
DANN | Uncertain | 1.0 | |
DEOGEN2 | Pathogenic | 0.99 | D |
Eigen | Uncertain | 0.56 | |
Eigen_PC | Uncertain | 0.43 | |
FATHMM_MKL | Uncertain | 0.92 | D |
FuncVEP CTI | Pathogenic | 1.0 | |
GPN-Star LLR | N/A | -8.7 | |
GPN-Star score | N/A | 8.7 | |
LIST_S2 | Uncertain | 0.94 | D |
M_CAP | Pathogenic | 0.93 | D |
MetaRNN | Pathogenic | 0.94 | D |
MetaSVM | Pathogenic | 1.1 | D |
Mutation Taster | N/A | 4/96 | disease causing (ClinVar) |
MutationAssessor | Pathogenic | 3.7 | H |
PhyloP100 | Uncertain | 3.8 | |
popEVE | Benign | -4.1 | |
PrimateAI | Pathogenic | 0.87 | D |
PROVEAN | Pathogenic | -6.8 | D |
REVEL | Pathogenic | 0.94 | |
Sift | Uncertain | 0.0060 | D |
Sift4G | Uncertain | 0.010 | D |
Varity_R | N/A | 0.99 | |
VESM-3B | Benign | -17 |
Splicing Scores
| Algorithm | Calibrated pred. | Score | Prediction |
|---|---|---|---|
Pangolin (max) | Benign | 0.0 | |
SpliceAI score (max) | Benign | 0.010 | Details are displayed if max score is > 0.2 |
Find out detailed SpliceAI scores and Pangolin per-transcript scores at
MaxEntScan Visualizer can be used to analyze the impact of this mutation on the neighboring sequence.